10725819
Kidney, hrr
https://raw.githubusercontent.com/jamesd4335/hrr2/master/hrr_pipes.csl
50
date
desc
197
https://healthresearchreport.com/wp-content/plugins/zotpress/
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Vitamin C-induced hyperoxaluria causing reversible tubulointerstitial nephritis and chronic renal failure: a case report##!2007 | Rathi S, Kern W, et al. | Journal of Medical Case Reports. 2007;1(1):155. ##!http://doi.org/10.1186/1752-1947-1-155
Ascorbate Increases Human Oxaluria and Kidney Stone Risk##!2005 | Massey LK, Liebman M, et al. | The Journal of Nutrition. 2005;135(7):1673-1677. ##!http://doi.org/10.1093/jn/135.7.1673
Acute Oxalate Nephropathy After Massive Ascorbic Acid Administration##!1985 | Lawton JM, Conway LT, et al. | Archives of Internal Medicine. 1985;145(5):950-951. ##!http://doi.org/10.1001/archinte.1985.00360050220044
Renal Failure Secondary to Massive Infusion of Vitamin C##!1984 | McAllister CJ, Scowden EB, et al. | JAMA. 1984;252(13):1684. ##!http://doi.org/10.1001/jama.1984.03350130016019
Acute Kidney Injury From Biopsy-Proven Renal Oxalosis From Excessive Intake of Vitamin C Leading to End-Stage Kidney Disease##!Williams R, Dauleh M, et al. | Cureus. 14(12):e33061. ##!http://doi.org/10.7759/cureus.33061